Forensic Aspects - Neural Functions



A nervous system performs many high-order functions that are not easily classified according to anatomical or chemical processes in which disorders tend to be recognized clinically in the form of a syndrome. Five main neural dysfunctions of complex origin and character to be discussed in this chapter are epilepsy and seizure disorders, the phenomenon of dementia, cognitive–perceptual deficiencies, behavioural illness, and disturbances of consciousness and coma. Five forms of complex neurological disease pose common difficulties for the forensic pathologist and neuropathologist because their pathology is complex or poorly understood, and there is often a great deal of difficulty in performing satisfactory clinical–pathological correlations using ordinary techniques. There ere are many forms of sudden attacks that manifest themselves as disorders of neurological function that are casually referred to as fits or seizures. These sudden, usually transitoryattacks may have many causes that do not immediately arise in the nervous system and may only involve it secondarily, as in the case of Stokes-Adams attacks and various syncopal attacks (cough syncope, micturitional syncope, etc, muscular disorders, and metabolic diseases such as diabetes. Some apparent epileptic seizures have no electrical basis and represent fictitious seizures or hysterical reactions. Classification of epileptic seizures is not universally agreed upon. Without discussing the merits of the various systems and the arguments for or against them, probably the most universally recognized and useful is the so-called clinical and electroencephalographic classification. According to a clinical EEG classification, there are four basic forms of epileptic seizures:

1. Partial seizures or seizures beginning locally
2. Generalized seizures, be they bilateral or symmetrical and without local onset
3. Unilateral or predominantly unilateral seizures
4. Otherwise unclassified seizures


Generalized seizures can be separated into so-called absence attacks (petit mal seizures) or several other forms enumerated below. An absence attacks can be simple or complex, involving not only staring but also autonomic, motor, postural, or other functional abnormalities. Other generalized seizures may be divided into myoclonic jerks, infantile spasms (hypsarrhythmia), clonic seizures, tonic seizures, generalized tonic–clonic (grandmal) seizures, or atonic–akinetic seizures. Unilateral or predominantly unilateral seizures constitute a mixed category in which whatever the symptomatology they show, it is primarily unilateral and not generalized. In the case of generalized tonic–clonic convulsions (GTCs; grand mal seizure), there may be no aura or warning of an attack. A classic GTC attack often begins with a loud cry, loss of consciousness, and tonic contraction of muscles leading to collapse, during which bladder and bowel control may be lost. Breathing may be suspended and cyanosis may occur. As the seizure ends, the patient may be drenched in sweat, be flaccid or atonic, and show Babinski signs. During the attack, individuals are unconscious and unable to perform any meaningful or integrated function, they are very vulnerable to accidents. They may fall and injure themselves, drop smoking materials and ignite a fire, or injure others by falling or violent movements. Partial or focal fits are typified by the classic Jacksonian seizure, which usually begins with involuntary spasm of a body part, usually an extremity (fingers, hand, toes) but it may include the mouth. Consciousness may be preserved as long as the seizure is localized but is usually lost during the generalized phase. Other forms of focal fits may affect specific functional regions of the brain, such as the temporal and limbic regions, speech centers, and visual or auditory centers. Occasionally, verbal automatisms occur, in which nonsensical words are spoken or profanities or abusive words or phrases are yelled. Sometimes these focal events include spitting, involuntary urination, or defecation. Many of these types of seizure symptoms are typical for so-called temporal lobe epilepsy or psychomotor epilepsy. Sometimes lip movements, grimacing, or small myoclonic jerks may accompany the fit.  A greater understanding of the organization of the cerebral cortex anatomically and physiologically can explain many forms of seizures, especially those in which there is structural damage or malformation of the cortex and subcortex. Recurrent collateral fibers coming from the cortex extend into the subcortex and then arborize upward into the cortex again to inhibit neural activity. Causes of epilepsy are numerous and include congenital malformations, perinatal brain injury, trauma, infection, metabolic abnormalities, vascular disease, hypoxia, electrolyte abnormalities and dehydration (hypernatremia, hyponatremia, hypocalcemia, hypomagnesemia), renal and hepatic failure, tumors, degenerative diseases, demyelinating diseases, alcohol and drug toxicity and withdrawal, hyperpyrexia, and various poisons. Some individuals experience increased seizure frequency during menstruation and in pregnancy, where some imbalance in fluid and electrolyte metabolism may be responsible but has not been proven. Some evidence exists that estrogen levels may play a role in seizure thresholds, and birth control pills have been the subject of some concern in this regard. Trauma is a common inducer of the epileptic state, both as a primary acute event and later as a secondary chronic event due to cortical contusion. Seizures of any clinical form, but which are usually generalized, that occur early after trauma often connote a more serious head injury with a much higher fatality rate than if seizures occur later. The mechanism of seizure production is not always clear but may result from deafferentation of cortical tissue or irritation of instability in viable cortical tissue hemorrhage.


Epilepsy is a significant public health problem in every country of the world, but precise and comprehensive figures on incidence are difficult to obtain because definitions of what constitutes epilepsy vary and methods for obtaining data are not uniform. Postmortem toxicological studies in this same group revealed that 70 to 80% of cases had either no detectable levels of anticonvulsants (phenobarbital or phenytoin) or subtherapeutic levels of these agents, whereas the remainder had therapeutic levels of at least one anticonvulsant at the time of death.

Acknowledgements:
The Police Department; 
www.politie.nl and a Chief Inspector – Mr. Erik Akerboom     ©

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